Blood thinners have become essential in our modern lives. Many of us carry the risk of cardiac arrests, precipitating the need for one. So how do they work?
Before we understand that, let us understand why they are so vital. Our blood is made up of plasma, platelets, red blood cells, and white blood cells, as an example.
Since we can bruise or cut ourselves, it is essential that our blood contains something to block the cut and prevent us from bleeding to death.
However, if our blood were to be too thick, it would not flow and move around our body, which is required to transport nutrients, oxygen, and other materials essential for our survival.
So, in a sense, the thickness or viscosity of our blood has to be in a narrow range.
One of the common problems with cardiovascular risk is that our blood gets thick. Blood clots then form, creating blockages that prevent blood from flowing.
If this were to happen to a critical artery, it could prevent blood from flowing to the heart or brain, causing a stroke. So using a blood thinner in such circumstances is essential.
There are different kinds of blood thinners.
An antiplatlet that prevents your body from creating clumps from platelets. You have probably heard of the phrase "an aspirin a day keeps the doctor away." The recommendation was made to prevent your blood from creating clots.
Aspirin or an equivalent, is more commonly taken by those who have already had a stroke. By the way, the debate is still open on whether aspirin is good or not for you.
An anticoagulant that slows down the process of making clots. In this case, you are trying to prevent the blood from thickening and creating lumps.
Naturally, neither comes without risks. Bleeding, constipation, red color urine are some of the possible side effects. As always, your Doctor would know best.
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